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Differential secretion of interleukin-8 by human epithelial cell lines upon entry of virulent or nonvirulent Yersinia enterocolitica.

polymorphonuclear leukocytes, upon bacterial entry. In this study we analyzed the secretion of IL-8 by human intestinal (T84) and cervical (HeLa) epithelial cell lines in response to infection with the enteric ...

Mapping of TH1 helper T-cell epitopes on major secreted mycobacterial antigen 85A in mice infected with live Mycobacterium bovis BCG36652

62, Issue 2, Number 370, p.363- 370 (1994) Keywords: 0 a Amino Acid Sequence an Animal Animals Antigens Antigens,Bacterial article AS BCG Belgium Beta biosynthesis Brussels Cell cells Class Cytokines ...

Characterization of structural and non-structural proteins of hog cholera virus by means of monoclonal antibodies.

Keywords: Animals Antibodies, Monoclonal Blotting, Western Cattle Cell Line Classical swine fever virus Cross Reactions Diarrhea Viruses, Bovine Viral Electrophoresis, Polyacrylamide Gel Epitopes ...

Comparative analysis of monoclonal antibodies against pestiviruses: report of an international workshop.

Microbiol, Volume 20, Issue 2, p.123-9 (1989) Keywords: Animals Antibodies, Monoclonal Antibodies, Viral Cell Line Cross Reactions Fluorescent Antibody Technique Immunoenzyme Techniques mice Pestivirus ...

Anchoring tick salivary anti-complement proteins IRAC I and IRAC II to membrane increases their immunogenicity.

Vet Res, Volume 40, Issue 5, p.51 (2009) Keywords: Animals Antibodies, Monoclonal Cats Cattle Cell Adhesion Cell Line Complement Inactivator Proteins Complement System Proteins Dogs Gene Expression ...

Genetic immunisation of cattle against bovine herpesvirus 1: glycoprotein gD confers higher protection than glycoprotein gC or tegument protein VP8.

Vet Res, Volume 36, Issue 4, p.529-44 (2005) Keywords: Animals Antibodies, Viral Capsid Proteins Cattle Cell Line Glycoproteins Herpesvirus 1, Bovine Herpesvirus Vaccines Time Factors Vaccines, DNA ...

RNA-based drug susceptibility testing of Mycobacterium tuberculosis

resistance mutations, (b) do not distinguish living from dead cells, (c) ignore all intrinsic resistance mechanisms, and (d) ignore the influence of compensatory mutations. Objectives We introduce ...

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